Santé bien être nutrition environnement / Health

It is a further and very worrying step down the wrong road for humanity.

• Obesity, especially increased belly fat, insulin resistance,
  and diabetes: diabetics have up to 65 percent higher risk of developing
  Alzheimer's disease. Keep your fasting insulin level below 3 by
  minimizing sugar and grains and exercising regularly.
  
• Elevated uric acid levels: fructose is one of the primary factors that drives up uric acid, which is another reason to avoid excess sugar.
  
• High blood pressure.
  
• Heart disease and atrial fibrillation^iv: If you have "atrial fib," you are 44 percent more likely to develop dementia.
  
• Thyroid dysfunction.^v
  
• Head trauma^vi:
  There appears to be a "strong link" between a history of head trauma
  and Alzheimer's disease, especially if the trauma occurred repeatedly or
  involved loss of consciousness—so keep your seat belt fastened.
  
• Anticholinergic drugs:
  Drugs that block acetylcholine, a nervous system neurotransmitter, have
  been shown to increase your risk of dementia. These drugs include
  certain nighttime pain relievers, antihistamines, sleep aids, certain
  antidepressants, medications to control incontinence, and certain
  narcotic pain relievers.One study^vii
  found that those who took drugs classified as "definite
  anticholinergics" had a four times higher incidence of cognitive
  impairment. Regularly taking two of these drugs further increased the risk.
  

• itamin B deficiencies: Elevated homocysteine levels due to vitamin B6, B12 and folate deficiencies; according to a small Finnish study recently published in the Journal Neurology^viii, people who consume foods rich in B12 may reduce their risk of Alzheimer's in their later years.
  
• Insufficient omega-3 fats:
  High intake of the omega-3 fatty acid DHA lowers your risk for
  Alzheimer's by preventing the associated cell damage. Researchers also
  report that DHA "dramatically reduces the impact of the Alzheimer's
  gene."
  
• Vitamin E deficiency^ix: A 2008 study showed that Alzheimer's patients who took 2,000 IU of vitamin E daily showed a 26 percent lower mortality rate.
  

• Aluminum toxicity: avoid antiperspirants, non-stick cookware, and vaccines containing aluminum (and mercury) adjuvants/additives, such as the flu vaccine
  
• Mercury
  toxicity: Avoid fish and seafood, which is usually contaminated with
  mercury, and avoid dental amalgams, which are probably the largest
  sources of mercury; implement the mercury detox protocol and utilize a biological dentist if you have dental amalgams
  
• Fluoride toxicity: More than 23 different studies indicate even moderate fluoride exposure may have damaging effects on your brain
  
• Copper toxicity:
  Evidence exists that copper can leach out of copper pipes and into tap
  water, and when consumed over time, can build up in your body. Copper
  toxicity has been linked to Alzheimer's disease, heart disease, and
  diabetes. You should also avoid vitamin/mineral pills that contain
  copper and iron, especially if you're over the age of 50
  
• Cell phone radiation.
  Radiation from cell phones and mobile phone handsets may damage areas
  of your brain associated with learning, memory and movement, and may
  trigger Alzheimer's disea
  

Finally, there are a few other nutritional recommendations worth
noting for their specific benefits in preventing and treating dementia.
So, although your fundamental strategy for preventing dementia should
involve a comprehensive lifestyle approach, you may want to consider
adding a few of these natural dietary agents to your anti-Alzheimer's
arsenal.

The three natural foods/supplements that have good science behind
them, in terms of preventing age-related cognitive changes, are coconut
oil, Gingko biloba, and alpha lipoic acid.

1. Coconut Oil: The
   primary fuel your brain needs for energy is glucose. However, your
   brain is able to run on more than a single type of fuel, one being
   ketones (ketone bodies), or ketoacids. Ketones are what your body produces when it converts fat (as opposed to glucose) into energy.The medium chain triglycerides (MCT) found in coconut oil are
   GREAT source of ketone bodies, because coconut oil is about 66 percent
   MCTs.In fact, ketones appear to be the preferred source of brain food in
   patients affected by diabetes or Alzheimer's.
   
2. Gingko biloba: Many
   scientific studies have found that Ginkgo biloba has positive effects
   for dementia. Gingko, which is derived from a tree native to Asia, has
   long been used medicinally in China and other countries.
   
   Fifteen years ago, in one of the first issues of my newsletter, I posted the results of a 1997 study from JAMA^x
   that showed clear evidence that Ginkgo improves cognitive performance
   and social functioning for those suffering from dementia. Research since
   then has been equally promising.
   
   One study in 2006 found Gingko as effective as the dementia drug
   Aricept (donepezil) for treating mild to moderate Alzheimer's type
   dementia. A 2010 meta-analysis^xi found Gingko biloba to be effective for a variety of types of dementia.
   
3. Alpha lipoic acid (ALA): ALA can stabilize cognitive functions among Alzheimer's patients and may slow the progression of the disease.
   

As it turns out, this type of genetic modification, called cytoplasmic
transfer, is actually a hot topic among geneticists, but it's rarely
published or discussed in the lay press, if at all—as evidenced by my
own surprise when reading this decade-old piece of news.

Many follow-up reports continue to tout the high success of this method
of treating infertility. But some, including a book put out by
Cambridge Press, warns of the dangers and risks of this procedure. For
example, the following excerpts from a report³ delivered during the 2003 World Congress on Controversies in Obstetrics, Gynecology & Infertility
in Berlin raises questions about the less than thoughtful
implementation of this technology, and some of the problems
encountered:

"... Cytoplasmic control of preimplantation development is not a
"new" concept, but ooplasm transfer have been amazingly rapidly applied
in humans, with relative success, in the absence of extensive research
to evaluate the efficacy and the potential risks of the method,
resulting in some publications highlighting the potential dangers
(Winston and Hardy 2002, DeRycke et al 2002, Templeton 2002), and
unpredictable outcomes (Cummins 2001, 2002).

... A frank follow-up of ooplasmic transplantation pregnancies and
infants reports that two out of 17 fetuses had an abnormal 45, XO
karyotype. The authors assume the hypothesis of a link between
chromosomal anomalies and oocytes manipulation, and reveal that one of
the babies has been diagnosed at 18 months with Pervasive Developmental
Disorder, a spectrum of autism-related diagnoses." [Emphasis mine]

So it didn't take long—less than two years, in fact—for reports of
"unpredictable outcomes" to crop up. I for one am not surprised. It's
somewhat disconcerting that so much of this research is taking place
without open discussion about the ethical questions associated with it.


The US FDA appears to have begun looking at the ethics of ooplasmic transplantation, and in one powerpoint⁴
it is pointed out that an 18-month-old child born from this procedure
has been diagnosed with autism (PDD), and that the incidence of
chromosomal anomalies is known to be higher in children born from the
procedure than the rate of major congenital abnormalities observed in
the natural population.

The document also states that lack of testing and long-term follow-up
of the children born from the procedure so far is a significant
shortcoming, making evaluation of the safety and effectiveness of the
technique very difficult. The genetic modification of humans appears to
have been running alongside the genetic engineering of plants, being
just a few years behind in terms of the technology being unleashed, and
the lack of proper evaluation of health effects is apparently on par
as well, which is to say near non-existent...

*** Comme le remarque le D^r
Catherine Thillier-Gasc, médecin à Angers et spécialiste de la
physiopathologie des perturbateurs endocriniens, il en va de même de
certaines associations anti-pesticides, qui sont toujours mobilisées
lorsqu’il s’agit d’affirmer que les pesticides sont responsables de
malformations génétiques, mais restent bien silencieuses au sujet de la
génistéine, sous prétexte que ce phyto-estrogène est naturel !

Références :
1. Mark Messina, « Soy Intake and Cancer Risk : A Review of the In Vitro
and In Vivo Data », Nutrition and Cancer, 1994.
2. N. L. Petrakis, et al, « Stimulatory influence of soy protein isolate
on breast secretion in pre-and postmenopausal women », Cancer Epid Bio
Prev,1996.
3. Craig Dees, et al, « Dietary estrogens stimulate human breast cells
to enter the cell cycle », Environmental Health Perspectives,1997.
4. R. L. Divi, et al, « Anti-thyroid isoflavones from the soybea »,
Biochemical Pharmacology, 1997.
5. Claude Hughes, « Soy Intake May Affect Fetus », Reuters News Service,
November 5, 1999.
6. « Vegetarian diet in pregnancy linked to birth defect », British
Journal of Urology International, January 2000.


Gil Rivière-Wekstein
Publié dans le Numéro d’A&E MAI 2006
http://www.agriculture-environnemen...

Je suis devenu diabétique à l’âge de vingt-cinq ans. La littérature
sur le diabète fait parfois référence à un lien possible entre
l’absorption de lait de vache et le diabète. Mais bien sûr, les médecins
s’occupent de traiter la maladie et ne s’intéressent pas à
l’alimentation. Si vous faisiez des tests cliniques pour prouver que le
lait de vache cause le diabète et que vous stoppiez le développement de
cette maladie, on vous décernerait le prix Nobel.

Leur conclusion montrait une relation absolue de cause à effet entre
la consommation de lait et le diabète, (Fava et al., "relation entre la
consommation de produits laitiers et l’apparition du diabète mellite
insulino-dépendant ("IDDM") chez l’enfant en Italie", Diabetes Care
[traitement du diabète] 17(12):1488-90, déc. 1994).

Cependant, le calcium dans le lait n’est pas absorbé de
manière appropriée et la consommation de lait est un suspect sur la
liste des causes de l’ostéoporose.
Encore plus effrayant est le fait que le lait est
considéré comme l’un des responsables de la raison pour laquelle une
femme Américaine sur six va développer un cancer du sein.